We are all intrigued by the famous Albert Einstein. We think that some thing about his brain must have been different from ours and thats why he was a genius and we are average people struggling to understand his theory of relativity.
We also know that Thomas Harvey stole his brain after his death, presumably to conduct research. And many resarchers have actually conducted research on the genius’ brain. Here are some findings that may explain why he was different from us.
1. Dr. Marian Diamond, who worked at the University of California at Berkeley, reported that Einstein did indeed have a higher ratio of glial cells to neurons than other brains, and she hypothesized that the number of glial cells increased because of the high metabolic demand that Einstein put on his neurons. She meant that because einstein used his brain so much, he had more glial cells. This is study has still not been replicated and other researchers cite reasons which may disprove this conclusion. So the jury is still out on this one.
2. In 1996, a University of Alabama researcher named Britt Anderson published another study on Einstein’s brain with much less hullaballoo. Anderson had discovered that Einstein’s frontal cortex was much thinner than normal, but that it was more densely packed with neurons.
3. Thomas Harvey also had researcher Dr. Sandra Witelson study the brain. She selected pieces of the temporal and parietal lobes, and she also pored over the photographs Harvey had commissioned of the brain at the time of Einstein’s death. She noticed that Einstein’s Sylvian fissure was largely absent. The Sylvian fissure separates the parietal lobe into two distinct compartments, and without this dividing line, Einstein’s parietal lobe was 15 percent wider than the average brain. Significantly, the parietal lobe is responsible for skills such as mathematical ability, spatial reasoning and three-dimensional visualization. This seemed to fit in perfectly with how Einstein described his own thought process: “Words do not seem to play any roles,” he once said. “[There are] more or less clear images”. Witelson hypothesizes that the lack of a Sylvian fissure may have allowed the brain cells to crowd in closer to one another, which in turn enabled them to communicate much faster than normal. This brain structure may also have had something to do with Einstein’s delayed speech developmen. At this point, scientists don’t know enough about how the brain works to know if Witelson’s work is accurate, but this is the accepted theory at the moment.